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Aspartic Acid

C4H7NO4 organic

Properties

StateSolid (white crystalline powder)
ColorWhite
SolubilitySlightly soluble in water (5.4 g/L at 25°C); soluble in dilute acids and bases
Melting Point270°C (decomposes)
Boiling PointDecomposes before boiling

About Aspartic Acid

Aspartic acid is one of the two acidic amino acids — the other is glutamic acid — and at physiological pH it exists almost entirely as the anionic aspartate, with the side-chain carboxyl deprotonated (pKa3 ≈ 3.65). Its compact four-carbon structure with carboxyls at both ends gives it an unusually central role in metabolism. Aspartate is one of the two nitrogen donors in the urea cycle (the other is ammonia), reacting with citrulline to form argininosuccinate and contributing the second nitrogen that ultimately ends up in urea. In nucleotide biosynthesis, aspartate is the source of the N1 atom in purines and contributes the entire pyrimidine ring carbon-nitrogen backbone — every cell synthesizing DNA needs aspartate continuously. As a residue in proteins, aspartate side chains often coordinate metal ions in active sites (the canonical aspartyl proteases, including HIV protease and renin, use two aspartate residues to activate water for amide-bond hydrolysis), and they participate in the catalytic triad of serine proteases like trypsin. The artificial sweetener aspartame is the methyl ester of the dipeptide L-aspartyl-L-phenylalanine, and roughly 200x sweeter than sucrose by mass — though metabolically the body hydrolyzes it back to its three components, including the aspartate.

Where you'll encounter it

If you've consumed any diet soda in the last forty years, you've consumed aspartame, which contains an aspartic acid residue that gets liberated by digestive proteases in your gut. In a biochemistry teaching lab, aspartate's role in the urea cycle is one of the canonical examples used to illustrate how amino-acid catabolism feeds nitrogen excretion: feed labeled aspartate to a hepatocyte preparation, follow the labeled nitrogen, and you'll see it appear in urea within minutes. In drug discovery, aspartyl-protease inhibitors are a major drug class — the HIV protease inhibitors (saquinavir, ritonavir, lopinavir) all target a pair of aspartate residues in the viral enzyme's active site, and the tactic is direct enough that the corresponding crystal structures appear in nearly every introductory medicinal-chemistry textbook.

Common Uses

  • Urea-cycle nitrogen donor for liver-based ammonia clearance
  • Building block for purine and pyrimidine nucleotide biosynthesis
  • Aspartame precursor in artificial-sweetener manufacture
  • Active-site residue in HIV protease and other aspartyl protease drug targets
  • Excitatory neurotransmitter in select brain regions

Safety Information

GRAS for dietary use; non-toxic at the levels found in normal food and supplements. The aspartame controversy — whether the methanol released during aspartame hydrolysis poses health risks — has been studied repeatedly, and major regulatory bodies (FDA, EFSA, JECFA) have consistently concluded the dietary exposure is well below toxicity thresholds. People with phenylketonuria (PKU) need to avoid aspartame, but the warning is for the phenylalanine component, not the aspartate.

This safety summary is for educational reference only and may not be complete. It is not a substitute for Safety Data Sheets (SDS), medical advice, or professional chemical safety guidance. Always consult appropriate SDS and qualified professionals before handling chemicals.

Constituent Elements

Frequently Asked Questions

What is the molar mass of aspartic acid?
133.103 g/mol. Sum 4(12.011) for the carbons, 7(1.008) for the hydrogens, 14.007 for the lone nitrogen, and 4(15.999) for the four oxygens, giving 133.10. The number is useful in nutritional biochemistry for converting between the daily aspartate intake (typically a few grams per day from dietary protein) and millimolar concentrations in plasma.
What role does aspartic acid play in the urea cycle?
Aspartate condenses with citrulline (catalyzed by argininosuccinate synthetase, with ATP as the energy source) to form argininosuccinate. The next enzyme, argininosuccinate lyase, splits that intermediate into arginine and fumarate — the arginine continues through the cycle to release urea, while the fumarate enters the citric acid cycle. The net effect is that aspartate has donated one of its nitrogens to urea while its carbon skeleton has been recycled through the TCA cycle as fumarate. The pathway is the reason that urea-cycle defects often present with elevated plasma aspartate.
How is aspartic acid different from asparagine?
Same backbone, different side chain. Aspartic acid carries a side-chain carboxyl (-CH2-COOH), which is negatively charged at physiological pH. Asparagine carries the corresponding amide (-CH2-CONH2), which is uncharged and acts as a hydrogen-bond donor and acceptor. Asparagine is biosynthesized from aspartate by asparagine synthetase, which transfers the amide nitrogen from glutamine to the aspartate side chain at the cost of an ATP. The two amino acids both appear in proteins but play different roles: aspartate's negative charge supports metal coordination and catalysis, asparagine's uncharged amide supports hydrogen-bonded recognition surfaces.