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Caffeine

C8H10N4O2 organic

Properties

StateSolid (white crystalline powder with bitter taste)
ColorWhite
SolubilityModerately soluble in water (22 g/L at 25°C); more soluble in hot water
Melting Point235°C (sublimes)
Boiling PointSublimes at 178°C

About Caffeine

Caffeine is 1,3,7-trimethylxanthine, a methylated purine alkaloid whose mechanism is one of the cleanest receptor-pharmacology stories in pharmacology. The molecule is a near-perfect mimic of adenosine at the A1 and A2A receptors — the bicyclic purine scaffold is shared, and the methylation pattern at N1, N3, and N7 occupies just enough of the binding pocket to act as a competitive antagonist without triggering downstream Gi signaling. When adenosine accumulates in the basal forebrain over a waking day and binds A2A receptors there, it inhibits the wake-promoting neurons that gate sleep onset. Caffeine displaces adenosine, and the disinhibition is what we feel as alertness. The Ki at human A2A is about 2.4 µM, which is comfortably below the 5–8 µM peak plasma concentration after a 200 mg dose, so a regular cup of coffee actually saturates a meaningful fraction of A2A receptors. The molar mass is 194.19 g/mol, the melting point is 235 °C with sublimation, and the 22 g/L water solubility at 25 °C jumps to 660 g/L in boiling water — which is why coffee and tea are made hot. Friedlieb Ferdinand Runge isolated caffeine from coffee beans in 1819 after Goethe handed him a box and asked him to figure out what the active principle was. Beyond beverages, caffeine is added to OTC analgesics like Excedrin and Anacin because it potentiates the analgesic effect of aspirin and acetaminophen by roughly 40% (probably through cerebral vasoconstriction and adenosine-receptor antagonism in pain pathways), and the caffeine-from-tea-leaves DCM extraction is the most-performed liquid-liquid extraction in undergraduate organic teaching labs.

Where you'll encounter it

If you've ever extracted caffeine from black tea in a sophomore orgo lab — boil tea bags in dilute Na2CO3, partition with dichloromethane in a separatory funnel, dry over anhydrous MgSO4, rotovap, sublime the off-white residue to crystalline white needles — you've worked with caffeine as the prototype natural-product extraction. In a clinical setting, IV caffeine citrate is the standard treatment for apnea of prematurity in NICU infants; the half-life in neonates is 100 hours versus 5 hours in adults because their CYP1A2 expression is still ramping up. In a sports-nutrition lab, caffeine pharmacokinetics matter for the WADA threshold (formerly 12 µg/mL in urine, now de-listed but still monitored) and for the well-replicated 2–6% endurance-performance benefit at 3–6 mg/kg doses.

Common Uses

  • Adenosine A1/A2A receptor antagonist in coffee, tea, and energy drinks
  • Analgesic adjuvant in OTC headache combinations (Excedrin, Anacin)
  • IV caffeine citrate for apnea of prematurity in neonatal intensive care
  • Standard liquid-liquid extraction target in undergraduate organic teaching labs
  • Ergogenic aid at 3–6 mg/kg for endurance-sport performance

Safety Information

Harmful if swallowed at high doses. GHS H302 (harmful if swallowed). The acute LD50 in humans is roughly 150–200 mg/kg — about 10 g for a 70 kg adult, equivalent to ~100 cups of brewed coffee, but as little as 1–2 teaspoons of pure caffeine powder. The FDA has issued specific warnings against consumer-grade pure caffeine powder after several teen deaths from miscalculated doses. Therapeutic and recreational doses (50–400 mg) cause sympathetic stimulation, mild tachycardia, increased gastric acid secretion, and diuresis; chronic intake produces tolerance via A2A receptor upregulation, and abrupt discontinuation produces a 12–48 hour withdrawal syndrome of headache, fatigue, and impaired concentration.

This safety summary is for educational reference only and may not be complete. It is not a substitute for Safety Data Sheets (SDS), medical advice, or professional chemical safety guidance. Always consult appropriate SDS and qualified professionals before handling chemicals.

Constituent Elements

Frequently Asked Questions

What is the molar mass of caffeine?
Caffeine (C8H10N4O2) has a molar mass of 194.19 g/mol — 8 × 12.011 (C) + 10 × 1.008 (H) + 4 × 14.007 (N) + 2 × 15.999 (O). The pKa values are 0.6 (protonated N9, in strong acid only) and 14 (effectively non-acidic), so at physiological pH caffeine is neutral and crosses the blood-brain barrier readily by passive diffusion.
How does caffeine keep you awake?
Caffeine is a competitive antagonist at adenosine A1 and A2A receptors with a Ki around 2.4 µM at human A2A. Adenosine accumulates extracellularly during waking hours as a byproduct of ATP hydrolysis in active neurons; binding A2A in the basal forebrain inhibits wake-promoting cholinergic neurons and lets sleep pressure build. Caffeine displaces adenosine from A2A, the wake-promoting neurons disinhibit, and you feel alert. A 200 mg dose gives 5–8 µM peak plasma concentration, well above the Ki, with a half-life of about 5 hours in healthy adults — which is why caffeine after 2 PM still measurably disrupts sleep latency.
How much caffeine is in a cup of coffee?
A standard 8 oz (240 mL) cup of drip-brewed coffee contains 95 mg of caffeine on average, but the range is wide: 60–200 mg depending on bean variety (robusta has roughly twice the caffeine of arabica), roast level (light roasts retain slightly more caffeine than dark, contrary to common belief — they just taste less bitter), grind size, and brew time. A 1 oz espresso shot is around 65 mg, an 8 oz cup of black tea is 47 mg, a 12 oz can of cola is 35 mg, and a 16 oz energy drink runs 150–300 mg.